这就是 S级吗？
[Log] Day 7
Overcast. Rain imminent again.
"The lock and the key are one."
If true, does breaking the lock also change the nature of the "key"?
Unknown.
But I must try.
Because the alternative—becoming a puppet for "The Terminal," initiating the "evolution" of all humanity—is a fate worse than death.
At least that is a choice I keep as my own.
"Memory is not stored, only inherited."
Perhaps genes are such a memory. Not recorded on a hard drive, not carved in stone, but silently passed on with each cell division.
Whose memory are the locks within me?
Whose fear, whose caution, whose “perfection” was coded into my very genes?
I have no answer.
Only this certainty: I will break it all.
One lock. One chain. One organ at a time.
[Log] Day 8
Target: Liver.
Preparation:
Blood substitute and IV equipment.
Waterproof sheeting, for containing potential bleeding.
Stimulants and epinephrine, for cardiac arrest.
Selected a minor branch of the hepatic portal vein
—this vein handles partial venous return for the liver; blockage causes local ischemic necrosis, but isn't immediately fatal.
Controlled necrosis is required.
Procedure:
First, use Arcana to form an ultra-fine ice needle, approx. 0.1mm in diameter.
Then, under internal guidance, navigate the needle from a brachial vein, following the bloodstream, past the subclavian vein, superior vena cava, right atrium...
Left ventricle… finally reaching the target branch of the hepatic portal vein.
Navigation duration: four minutes.
Absolute focus required. Any error could cause the needle to block a critical vessel.
Upon reaching the target, hold position.
The cold begins affecting surrounding blood, forming micro-thrombi.
The thrombi occlude the vessel.
Liver tissue begins to ischemia.
Dull, diffuse pain in the right upper quadrant.
After five minutes of ischemia, as hepatocytes begin to die, the surrounding "locks" develop fine cracks.
Not shattered, just cracks.
But it's enough.
Immediately initiate repair:
Ice filaments flood in, first melting the thrombi, restoring flow. Then they adhere to the necrotic cells, isolating them.
Finally, a strange biological process begins—the translucent crystalline tissue starts "consuming" the dead cells, incorporating them.
Total duration: three hours.
Liver function remains largely normal.
The cracks in the locks haven't widened much, but they are there.
[Log] Day 9
Remembered the Master's words today:
"A killer's most precious quality isn't cruelty, nor skill, but decisiveness. The decisiveness to make a choice in the moment between life and death, and bear the consequences."
[Log] Day 10
Attempted to guide ice filaments to construct a brand-new, bionic structure in place of the frozen kidney.
This structure is not a true kidney. It lacks nephrons, lacks a complex filtration system.
But it has a "capillary network" of ice crystals that can simulate partial filtration functions. It has a semi-permeable membrane structure that selectively allows metabolic waste to pass, then centrally "freezes" it for storage.
Essentially, it's a built-in dialysis device. It can clear urea, creatinine, etc. Efficiency may be lower than a real kidney, but sufficient to sustain life.
More importantly:
The locks around the kidney are completely gone.
Not cracked, but gone.
Complete control is achieved in that region.
The cost: permanently losing a real organ.
[Log] Day 15
Can precisely perceive the status of every major organ.
Can slightly adjust heart rate, respiratory frequency via will.
Can control the secretion of certain hormones (e.g., adrenaline).
Can locally reduce pain signal transmission when injured.
These abilities are faint, elementary, but undeniably present.
"S-Class"
It's not merely increased energy output, but deep mastery over one's own life systems.
—
It is evolution.
[Log] Day 20
Two things happened today.
The first: the cat arrived.
I was adjusting the parameters of the crystalline renal construct late in the rain when a faint mewling sound pierced the downpour from the courtyard.
A Siamese cat.
Crouched under the wisteria trellis, utterly drenched, shivering.
Thin. Something wrong with its eyes.
—Pupils a pale, lilac-tinged violet, utterly unfocused.
Likely congenital cataracts or optic nerve hypoplasia from albinism.
Violet eyes too—but a deeper shade, like woodland violets.
I took it in.
No experience with pets, but the Master once said:
In extremis, any life can become an anchor.
The second breakthrough was intelligence-related.
The seventh attempt to breach the Genesis Threshold's external medical database.
This time, I shifted tactics. Instead of directly querying S's records, I searched for genetic research related to hyperalgesia.
The results were… unexpectedly successful.
I located an encrypted folder titled:
[Special Receptor Modulation Project – Subject S-7]
It took three hours to methodically crack six layers of encryption.
Inside was a complete medical dossier:
Name: S / Santali (Subject Designation S-7)
Genetic Profile:
Persistent upregulation in nociception-related gene expression:
?? TRPV1 (thermal/chemical nociception): +37%
?? TRPA1 (cold/mechanical nociception): +42%
?? P2X3 (tissue damage signal amplification): sensitivity increased by 89%
Inference: The sensory system remains in a prolonged "pre-activated" state, where external stimuli are easily amplified into pain signals.
Clinical Presentation:
Generalized hyperalgesia.
Low temperatures (≤5°C) trigger acute pain responses (confirmed by threshold testing).
Any reparative inflammatory process is accompanied by hypersensitive pain (indicative of central sensitization).
Observations:
?? Pain thresholds are markedly below baseline (mechanical, thermal, chemical).
?? Temperatures below 5°C induce sharp, persistent neuropathic pain (suspected TRPA1-mediated).
?? Abnormally enhanced pain feedback during tissue repair, consistent with neuropathic pain characteristics.
Treatment History:
Regimen: Long-term oral administration of a μ-opioid receptor inverse agonist (naloxone derivative, model NK-7α), with dosage escalated according to metabolic demand.
Mechanism: Antagonism of the endogenous opioid peptide system in an attempt to reset baseline pain perception.
Discontinuation Record: Treatment was halted six months ago.
Reason: Laboratory monitoring revealed that drug metabolites induced progressive hepatocellular vacuolation. The cumulative risk of drug-induced liver injury was assessed to outweigh clinical benefits.
Prognosis Note: Following discontinuation, pain sensitization may rebound. Neuroadaptive retraining is recommended.
·137% .
[Log] Day 25
Simultaneous ischemia in both lungs would induce systemic hypoxia. The brain's tolerance window is approximately four minutes. Beyond that, irreversible damage sets in.
The timing must be exact.
[Log] Day 29
Success.
The cat stayed throughout these three days. A silent witness.
[Log] Day 30
The cardiac attempt commences in one hour.
Final protocol:
Navigate an ice-needle with precision to the atrioventricular node—release just enough cryogenic energy to temporarily suspend the conduction cells' function without causing cellular death.
The freeze will auto-reverse after the predetermined interval: thirty seconds.
Calculated success probability: 40%.
[Log] Day 39
The rainy season has finally ended.
Success. It triggered a chain reaction.
The Locks—they've begun collapsing in earnest, like dominoes.
[Log] Day 55
Autumn rain.
The cat sleeps at my feet.
A strange quiet.
[Log] Day 81
A flower. Forged from ice, yet a flower in truth.
—has distinct venation, minute filaments in the pistil, and, in sunlight, it even demonstrates a faint form of transpiration, beading with microscopic droplets on its surface.
And it is violet. The quiet, deepening violet.
Is this... S-Class?